Relations of mitochondrial genetic variants to measures of vascular function

Jessica L. Fetterman, Chunyu Liu, Gary F. Mitchell, Ramachandran S. Vasan, Emelia J. Benjamin, Joseph A. Vita, Naomi M. Hamburg, Daniel Levy

Resultado de la investigación: Articlerevisión exhaustiva

4 Citas (Scopus)

Resumen

Mitochondrial genetic variation with resultant alterations in oxidative phosphorylation may influence vascular function and contribute to cardiovascular disease susceptibility. We assessed relations of peptide-encoding variants in the mitochondrial genome with measures of vascular function in Framingham Heart Study participants. Of 258 variants assessed, 40 were predicted to have functional consequences by bioinformatics programs. A maternal pattern of heritability was estimated to contribute to the variability of aortic stiffness. A putative association with a microvascular function measure was identified that requires replication. The methods we have developed can be applied to assess the relations of mitochondrial genetic variation to other phenotypes.

Idioma originalEnglish (US)
Páginas (desde-hasta)51-57
Número de páginas7
PublicaciónMitochondrion
Volumen40
DOI
EstadoPublished - may 2018
Publicado de forma externa

ASJC Scopus subject areas

  • Molecular Medicine
  • Molecular Biology
  • Cell Biology

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