Peripheral levels of sST2 are increased in patients with temporal lobe epilepsy: Additional evidence of low-grade inflammation

Eliana Cristina de Brito Toscano, João Marcelo Korcsik Lessa, Guilherme Nogueira de Oliveira, Ana Paula Gonçalves, Érica Leandro Marciano Vieira, Milene Alvarenga Rachid, Antônio Lúcio Teixeira

Producción científica: Articlerevisión exhaustiva

2 Citas (Scopus)

Resumen

Inflammation plays a pivotal role in temporal lobe epilepsy (TLE) pathophysiology. IL-33 can act as a transcription factor or as a cytokine, the latter through the transmembrane ST2 receptor or its soluble isoform (sST2), presenting a dual role in neurological diseases. The aim of this study was to determine the plasma levels of IL-33 and sST2 in parallel with clinical features in patients with TLE. Peripheral blood from patients and controls was sampled for the measurement of plasma levels of IL-33 and sST2 by enzyme-linked immunoassay (ELISA). While there were similar levels of IL-33 between controls and patients, sST2 were increased in patients. IL33 and sST2 plasma levels were not associated with TLE-related clinical features. In a subgroup analysis, IL-33 levels correlated with memory performance. In conclusion, our results reinforce the concept of chronic low-grade inflammation in patients with TLE.

Idioma originalEnglish (US)
Número de artículo107351
PublicaciónEpilepsy and Behavior
Volumen112
DOI
EstadoPublished - nov 2020
Publicado de forma externa

ASJC Scopus subject areas

  • Neurology
  • Clinical Neurology
  • Behavioral Neuroscience

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