Obesity enhances nongenomic estrogen receptor crosstalk with the PI3K/Akt and MAPK pathways to promote in vitro measures of breast cancer progression

Medicine and Dentistry

• Estrogen Receptor Alpha 100%
• Serum 81%
• Breast Cancer 72%
• Cell Growth 63%
• Cell Viability 63%
• Obesity 45%
• Patient 45%
• Cancer Cell 36%
• Mitogen Activated Protein Kinase 3 27%
• Mitogen-Activated Protein Kinase 27%
• Signal Transduction 18%
• Cancer Growth 18%
• Hormone Therapy 18%
• Akt Signaling 18%
• Therapy Resistance 18%
• Alpha Wave 18%
• Cells 18%
• Blood 18%
• MCF-7 9%
• In Vitro 9%
• Estrogen Receptor 9%
• Analysis of Variance 9%
• Body Mass Index 9%
• Student T Test 9%
• Postmenopause 9%
• Cell Signaling Pathway 9%
• Somatomedin C Receptor 9%
• Cancer Prognosis 9%
• Colony Formation 9%
• Introspection 9%
• Growth 9%
• MAPK Signaling 9%
• Assay 9%
• Targeted Therapy 9%
• Inpatient 9%

Biochemistry, Genetics and Molecular Biology

• Estrogen Receptor Alpha 100%
• Cell Growth 63%
• Cell Viability 63%
• Cancer Cell 36%
• Signal Transduction 27%
• Mitogen-Activated Protein Kinase 27%
• Akt Signaling 18%
• Alpha Wave 18%
• Clinical Study 18%
• Insulin-Like Growth Factor 1 Receptor 9%
• Body Mass 9%
• Estrogen Receptor 9%
• Colony Formation 9%
• Student's t-Test 9%
• Postmenopause 9%
• MAPK Signaling 9%
• Growth 9%
• Reduction (Chemistry) 9%
• Introspection 9%
• Serum (Blood) 9%