Elevated inflammatory response in caveolin-1-deficient mice with Pseudomonas aeruginosa infection is mediated by STAT3 protein and Nuclear Factor κB (NF-κB)

Medicine and Dentistry

• Alternative Complement Pathway C3 C5 Convertase 83%
• Aptitude 16%
• Caveolae 16%
• Caveolin 1 100%
• Cell Culture 16%
• Cytokine 16%
• Cytokine Response 16%
• Disease Exacerbation 16%
• Host Resistance 33%
• Infection 33%
• Interleukin 12p35 16%
• Kidney 16%
• Knock Out 50%
• Liver 16%
• Lung 16%
• Lung Injury 16%
• Macrophage 16%
• Membrane Invagination 16%
• Mortality 33%
• Nuclear Factor 83%
• Observation 16%
• Pathogen 16%
• Phenotype 16%
• Protein Analysis 16%
• Pseudomonas aeruginosa 33%
• Pseudomonas Infection 16%
• Respiratory Tract Infection 16%
• Serositis 33%
• Signal Transduction 16%
• STAT3 Protein 16%
• Superoxide 16%

Immunology and Microbiology

• Aptitude 16%
• Caveola 16%
• Cell Culture 16%
• Complement Factor B 83%
• Host Resistance 33%
• Infectious Agent 16%
• Interleukin 12p35 16%
• Kidney 16%
• Knockout Mouse 50%
• Liver 16%
• Lung 33%
• Membrane Invagination 16%
• Mortality 33%
• Mouse 16%
• Phenotype 16%
• Pseudomonas aeruginosa 50%
• Signal Transduction 16%
• STAT3 83%
• Superoxide 16%
• Tumor Necrosis Factor 50%
• Wild Type 16%

Biochemistry, Genetics and Molecular Biology

• Aptitude 16%
• Caveolae 16%
• Caveolin 1 100%
• Cell Culture 16%
• Cytokine Response 16%
• Host Resistance 33%
• Infectious Agent 16%
• Interleukin-6 50%
• Knockout Mouse 50%
• Membrane Invagination 16%
• Mortality 33%
• Mouse 16%
• Phenotype 16%
• Protein 16%
• Pseudomonas aeruginosa 50%
• Signal Transduction 16%
• STAT3 83%
• Wild Type 16%