An analog of withaferin a activates the MAPK and glutathione "stress" pathways and inhibits pancreatic cancer cell proliferation

Xiaobing Liu, Wenqing Qi, Laurence S. Cooke, E. M. Kithsiri Wijeratne, Ya Ming Xu, Marilyn T. Marron, A. A. Leslie Gunatilaka, Daruka Mahadevan

Producción científica: Articlerevisión exhaustiva

12 Citas (Scopus)

Resumen

Withaferin A (WA) (1) and two analogs [4-epi-withaferin A (2) and 4,27-diacetyl-4-epi-withaferin A (3)] were evaluated for antitumor activity in pancreatic cancer cells. IC50 for 1, 2, and 3 were 0.87, 0.45, and 0.29 μM (BxPC-3); 1.28, 1.53, and 0.52 μM (MIAPaCa-2); and 0.59, 2.25, and 0.56 μM (PANC-1), respectively. We chose WA analog 3 for functional studies with confirmatory RT-PCR and Western blotting. ANOVA identified 33 (MIAPaCa-2), 54 (PANC-1), and 48 (BxPC-3) gene expression changes. Fisher exact test demonstrated MAPK and glutathione pathways to be overexpressed with WA analog 3. WA analog 3 elicits a dose-and time-dependent apoptosis, activates MAPK and glutathione "stress" pathways, and inhibits proliferation.

Idioma originalEnglish (US)
Páginas (desde-hasta)668-675
Número de páginas8
PublicaciónCancer Investigation
Volumen29
N.º10
DOI
EstadoPublished - nov 15 2011
Publicado de forma externa

ASJC Scopus subject areas

  • Oncology
  • Cancer Research

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