Abstract
Traditionally it has been sait that transluminal angioplasty increases lumen diameter by compression and remodeling of atheromatous material. Recently, a new concept explaining the mechanics of angioplasty was described which challenges the classic concept of Dotter. It was argued that arterial balloon dilatation is achieved by intimal disruption and overstretching of the arterial wall, not by remodeling of atheromatous material.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 195-205 |
| Number of pages | 11 |
| Journal | Angiology |
| Volume | 35 |
| Issue number | 4 |
| State | Published - 1984 |
| Externally published | Yes |
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ASJC Scopus subject areas
- Cardiology and Cardiovascular Medicine
Cite this
The pathologic basis of angioplasty. / Castaneda-Zuniga, W. R.; Sibley, R.; Amplatz, K.
In: Angiology, Vol. 35, No. 4, 1984, p. 195-205.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - The pathologic basis of angioplasty
AU - Castaneda-Zuniga, W. R.
AU - Sibley, R.
AU - Amplatz, K.
PY - 1984
Y1 - 1984
N2 - Traditionally it has been sait that transluminal angioplasty increases lumen diameter by compression and remodeling of atheromatous material. Recently, a new concept explaining the mechanics of angioplasty was described which challenges the classic concept of Dotter. It was argued that arterial balloon dilatation is achieved by intimal disruption and overstretching of the arterial wall, not by remodeling of atheromatous material.
AB - Traditionally it has been sait that transluminal angioplasty increases lumen diameter by compression and remodeling of atheromatous material. Recently, a new concept explaining the mechanics of angioplasty was described which challenges the classic concept of Dotter. It was argued that arterial balloon dilatation is achieved by intimal disruption and overstretching of the arterial wall, not by remodeling of atheromatous material.
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UR - http://www.scopus.com/inward/citedby.url?scp=0021167635&partnerID=8YFLogxK
M3 - Article
C2 - 6231867
AN - SCOPUS:0021167635
VL - 35
SP - 195
EP - 205
JO - Angiology
JF - Angiology
SN - 0003-3197
IS - 4
ER -