Abstract
The cytadhesin (P1) structural gene of a spontaneous mutant of Mycoplasma pneumoniae which displayed a P1-negative phenotype was analyzed. An extra adenine was discovered in a stretch of normally seven adenines near the N-terminal region of the mutant P1 structural gene. The frameshift mutation resulted in the early termination of protein translation. Possible causes of the mutation are discussed.
Original language | English (US) |
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Pages (from-to) | 3237-3239 |
Number of pages | 3 |
Journal | Infection and immunity |
Volume | 57 |
Issue number | 10 |
State | Published - 1989 |
ASJC Scopus subject areas
- Parasitology
- Microbiology
- Immunology
- Infectious Diseases