Rapid onset of emphysema associated with diffuse parenchymal disease

Clarence A. Guenter; David A. Hanley; Brian J. Sproule; Jacqueline J. Coalson

doi:10.1016/0002-9343(79)90410-8

Rapid onset of emphysema associated with diffuse parenchymal disease

Clarence A. Guenter, David A. Hanley, Brian J. Sproule, Jacqueline J. Coalson

Pathology

Research output: Contribution to journal › Article › peer-review

1 Scopus citations

Abstract

In experimental models pulmonary emphysema may be produced in hours to days; however, in human subjects emphysema commonly develops over a period of many years. In this report we document a case of severe emphysema which developed in less than six months in association with an unexplained illness characterized by dyspnea, hypoxemia and bilateral lung parenchymal disease. There was no hereditary predisposition. The diffuse alveolar injury syndrome must be considered as a potential cause of alveolar disruption.

Original language	English (US)
Pages (from-to)	335-338
Number of pages	4
Journal	The American Journal of Medicine
Volume	67
Issue number	2
DOIs	https://doi.org/10.1016/0002-9343(79)90410-8
State	Published - Aug 1979

ASJC Scopus subject areas

Medicine(all)

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abstract = "In experimental models pulmonary emphysema may be produced in hours to days; however, in human subjects emphysema commonly develops over a period of many years. In this report we document a case of severe emphysema which developed in less than six months in association with an unexplained illness characterized by dyspnea, hypoxemia and bilateral lung parenchymal disease. There was no hereditary predisposition. The diffuse alveolar injury syndrome must be considered as a potential cause of alveolar disruption.",

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AB - In experimental models pulmonary emphysema may be produced in hours to days; however, in human subjects emphysema commonly develops over a period of many years. In this report we document a case of severe emphysema which developed in less than six months in association with an unexplained illness characterized by dyspnea, hypoxemia and bilateral lung parenchymal disease. There was no hereditary predisposition. The diffuse alveolar injury syndrome must be considered as a potential cause of alveolar disruption.

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