The traditional view that blood pressure (BP) can fundamentally be viewed as a function of cardiac output and systemic vascular resistance remains valid, although much has been learned about the many ways in which these two arms of the equation can be affected, down to the cellular level. The goal of this chapter is to provide the reader with an overview of the many pathophysiologic alterations that can lead to the development of hypertension (HTN).
|Original language||English (US)|
|Title of host publication||Pediatric Nephrology, Seventh Edition|
|Publisher||Springer Berlin Heidelberg|
|Number of pages||45|
|State||Published - Jan 1 2015|
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