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Overexpression of the α(1B)-adrenergic receptor causes apoptotic neurodegeneration: Multiple system atrophy
Michael J. Zuscik
, Scott Sands
, Sean A. Ross
, David J.J. Waugh
, Robert J. Gaivin
,
David Morilak
, Dianne M. Perez
Department of Pharmacology
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Article
›
peer-review
128
Scopus citations
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Medicine and Dentistry
Transgenic Mouse
100%
Neurodegeneration
100%
Brain
100%
Diseases
75%
Shy-Drager Syndrome
75%
Antagonist
50%
Central Nervous System
50%
Substantia nigra
50%
Age
50%
Cerebral Cortex
50%
Receptor
25%
Degeneration
25%
Etiology
25%
Phenotype
25%
Dysplasia
25%
Dopamine Agonist
25%
Tonic-Clonic Seizure
25%
Nerve Cell Degeneration
25%
DOPA
25%
Agonist
25%
Terazosin
25%
Tissues
25%
Pathology
25%
Epileptic Seizure
25%
Neuroscience
Neurodegeneration
100%
Multiple System Atrophy
50%
Eicosanoid Receptor
25%
Agonist
25%
Phenotype
25%
Dysplasia
25%
Dopaminergic
25%
Nerve Cell Degeneration
25%
Seizure
25%
Shy-Drager Syndrome
25%
Terazosin
25%
DOPA
25%
Epilepsy
25%