Neurosurgery for Cranial Dural Arteriovenous Fistulas Dural arteriovenous fistulae (DAVF)

Cameron M McDougall; Michael T. Lawton

doi:10.1007/978-3-642-37078-6_105

Neurosurgery for Cranial Dural Arteriovenous Fistulas Dural arteriovenous fistulae (DAVF)

Cameron M McDougall, Michael T. Lawton

Research output: Chapter in Book/Report/Conference proceeding › Chapter

1 Scopus citations

Abstract

Dural arteriovenous fistulae (DAVFs) are a subset of vascular lesions comprising 10–15 % of all cerebral vascular malformations. They are high-flow, low-resistance connections between a dural artery (or arteries) and a venous sinus or cortical vein. The fistulae lead to venous hypertension, creating ischemic conditions that can trigger a cascade of angiogenesis encouraging fistulous development in a vicious cycle. Cortical venous drainage of DAVFs increases their risk of hemorrhage or leads to nonhemorrhagic neurological deficits and necessitates treatment. DAVFs are treated most commonly through endovascular approaches, but some lesions are better treated with open surgical disconnection.

Original language	English (US)
Title of host publication	PanVascular Medicine, Second Edition
Publisher	Springer Berlin Heidelberg
Pages	2917-2941
Number of pages	25
ISBN (Electronic)	9783642370786
ISBN (Print)	9783642370779
DOIs	https://doi.org/10.1007/978-3-642-37078-6_105
State	Published - Jan 1 2015
Externally published	Yes

ASJC Scopus subject areas

Medicine(all)
Biochemistry, Genetics and Molecular Biology(all)

Access to Document

10.1007/978-3-642-37078-6_105

Fingerprint Dive into the research topics of 'Neurosurgery for Cranial Dural Arteriovenous Fistulas Dural arteriovenous fistulae (DAVF)'. Together they form a unique fingerprint.

Cite this

@inbook{1899691279334eccb09a3cce377991df,

title = "Neurosurgery for Cranial Dural Arteriovenous Fistulas Dural arteriovenous fistulae (DAVF)",

abstract = "Dural arteriovenous fistulae (DAVFs) are a subset of vascular lesions comprising 10–15 % of all cerebral vascular malformations. They are high-flow, low-resistance connections between a dural artery (or arteries) and a venous sinus or cortical vein. The fistulae lead to venous hypertension, creating ischemic conditions that can trigger a cascade of angiogenesis encouraging fistulous development in a vicious cycle. Cortical venous drainage of DAVFs increases their risk of hemorrhage or leads to nonhemorrhagic neurological deficits and necessitates treatment. DAVFs are treated most commonly through endovascular approaches, but some lesions are better treated with open surgical disconnection.",

author = "McDougall, {Cameron M} and Lawton, {Michael T.}",

year = "2015",

month = jan,

day = "1",

doi = "10.1007/978-3-642-37078-6_105",

language = "English (US)",

isbn = "9783642370779",

pages = "2917--2941",

booktitle = "PanVascular Medicine, Second Edition",

publisher = "Springer Berlin Heidelberg",

}

TY - CHAP

T1 - Neurosurgery for Cranial Dural Arteriovenous Fistulas Dural arteriovenous fistulae (DAVF)

AU - McDougall, Cameron M

AU - Lawton, Michael T.

PY - 2015/1/1

Y1 - 2015/1/1

N2 - Dural arteriovenous fistulae (DAVFs) are a subset of vascular lesions comprising 10–15 % of all cerebral vascular malformations. They are high-flow, low-resistance connections between a dural artery (or arteries) and a venous sinus or cortical vein. The fistulae lead to venous hypertension, creating ischemic conditions that can trigger a cascade of angiogenesis encouraging fistulous development in a vicious cycle. Cortical venous drainage of DAVFs increases their risk of hemorrhage or leads to nonhemorrhagic neurological deficits and necessitates treatment. DAVFs are treated most commonly through endovascular approaches, but some lesions are better treated with open surgical disconnection.

AB - Dural arteriovenous fistulae (DAVFs) are a subset of vascular lesions comprising 10–15 % of all cerebral vascular malformations. They are high-flow, low-resistance connections between a dural artery (or arteries) and a venous sinus or cortical vein. The fistulae lead to venous hypertension, creating ischemic conditions that can trigger a cascade of angiogenesis encouraging fistulous development in a vicious cycle. Cortical venous drainage of DAVFs increases their risk of hemorrhage or leads to nonhemorrhagic neurological deficits and necessitates treatment. DAVFs are treated most commonly through endovascular approaches, but some lesions are better treated with open surgical disconnection.

UR - http://www.scopus.com/inward/record.url?scp=84957003598&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=84957003598&partnerID=8YFLogxK

U2 - 10.1007/978-3-642-37078-6_105

DO - 10.1007/978-3-642-37078-6_105

M3 - Chapter

AN - SCOPUS:84957003598

SN - 9783642370779

SP - 2917

EP - 2941

BT - PanVascular Medicine, Second Edition

PB - Springer Berlin Heidelberg

ER -