Skip to main navigation
Skip to search
Skip to main content
Scholars @ UT Health San Antonio Home
Help & FAQ
English
Español
Home
Profiles
Research units
Publications
Datasets
Search by expertise, name or affiliation
Insulin resistance in uremia mediated by postbinding defects
D. Smith,
R. A. DeFronzo
Research output
:
Contribution to journal
›
Article
›
peer-review
109
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Insulin resistance in uremia mediated by postbinding defects'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Biochemistry, Genetics and Molecular Biology
Insulin
100%
Insulin Blood Level
66%
Insulin Binding
66%
Glucose
55%
Insulin Resistance
55%
Insulin Level
44%
Glucose Transport
44%
Gluconeogenesis
33%
Steady State
22%
Dose Response
22%
Glucose Metabolism
22%
Glucose Blood Level
11%
Monocyte
11%
Biological Activity
11%
Nursing and Health Professions
Insulin
88%
Control
77%
Insulin Blood Level
66%
Insulin Resistance
55%
Insulin Level
44%
Glucose
33%
Uremia
33%
Clamp
22%
Glucose Infusion
22%
Glucose Blood Level
11%
Receptor
11%
Infusion Rate
11%
Chronic Kidney Failure
11%
Procedures
11%
Receptor Reserve
11%
Tritium
11%
Insulin Infusion
11%
Iodine-125
11%
Medicine and Dentistry
Insulin Binding
66%
Insulin Resistance
55%
Glucose Uptake
44%
Glucose
33%
Gluconeogenesis
33%
Uremia
33%
Steady State
22%
Dose Response
22%
Glucose Metabolism
22%
Glucose Infusion
22%
Receptor
11%
Iodine 125
11%
Biological Activity
11%
Monocyte
11%
Chronic Kidney Disease
11%
Insulin Infusion
11%
Infusion Rate
11%
Tritium
11%
Receptor Reserve
11%
Neuroscience
Insulin
100%
Insulin Binding
66%
Glucose
55%
Glucose Transport
44%
Eicosanoid Receptor
11%
In Vivo
11%
Monocyte
11%
Receptor Reserve
11%
Suppression
11%