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High d(+)glucose concentration inhibits RANKL-induced osteoclastogenesis
Y. Wittrant
, Y. Gorin
, K. Woodruff
, D. Horn
, H. E. Abboud
, S. Mohan
, S. L. Abboud-Werner
Department of Medicine
Division of Nephrology/Renal Diseases
Department of Pathology
Research output
:
Contribution to journal
›
Article
›
peer-review
144
Scopus citations
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Medicine and Dentistry
Osteoclastogenesis
100%
Glucose
87%
Osteoclast Differentiation Factor
50%
Diabetes
37%
Hyperglycemia
25%
Biological Activity
25%
Nuclear Factor
25%
Bone Disease
25%
Caspase 3
25%
Cells
25%
Assay
25%
Reactive Oxygen Species
25%
Bone Marrow
25%
Complication
12%
Bone Turnover
12%
Chronic Disease
12%
Fracture
12%
Antioxidant
12%
Pathogenesis
12%
Metabolism
12%
Cell Function
12%
Diabetes Mellitus
12%
Bone Metabolism
12%
Osteopenia
12%
Introspection
12%
Levo Glucose
12%
Acid Phosphatase Tartrate Resistant Isoenzyme
12%
Calcitonin Receptor
12%
Dichlorodihydrofluorescein Diacetate
12%
Cathepsin K
12%
Comprehension
12%
Culture
12%
Macrophage
12%
Osteoporosis
12%
Biochemistry, Genetics and Molecular Biology
Osteoclastogenesis
100%
Glucose
75%
RANKL
50%
Hyperglycemia
25%
Glucose Level
25%
Biological Activity
25%
Caspase 3
25%
Species
25%
Bone Turnover
12%
Tartrate-Resistant Acid Phosphatase
12%
Fluorescence
12%
Cell Function
12%
Metabolic Pathway
12%
Calcitonin Receptor
12%
Cathepsin K
12%
Introspection
12%
Comprehension
12%
Immunology and Microbiology
Osteoclastogenesis
100%
RANKL
50%
Biological Activity
25%
Glucose Level
25%
Species
25%
Cells
25%
Bone
25%
Fluorescence
12%
Metabolism
12%
Cell Function
12%
Bone Metabolism
12%
Bone Turnover
12%
Introspection
12%
Comprehension
12%