Aspectos endocrinos, metabólicos y genéticos de la disfunción endotelial y su relación con la obesidad visceral

Translated title of the contribution: Endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation to visceral obesity

Raúl A. Bastarrachea Sosa, G. Castro Martínez, H. Laviada Molina, R. Yza Villanueva, G. Llamas Esperón, A. G. Comuzzie

    Research output: Contribution to journalReview articlepeer-review

    Abstract

    Atherosclerosis is the combination of pathological changes that not only affect cells deposited along the walls of the arteries, but also the inflammatory ones. Stimulus implied in the cause of atherosclerosis, which include LDL, mechanical forces followed by high blood pressure, and inflammatory response induced by metabolic and hemodynamic aspects as risk factors, are diverse. Resistance to insulin, followed by visceral obesity and diabetes mellitus type 2 represent equally chronic and complex systemic alterations. Thus, it is not strange that the intersection of these disorders and their final outcomes cause atherosclerotic diabetes. This article reviews the endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation with visceral obesity.

    Translated title of the contributionEndocrine, metabolic and genetic aspects of endothelial dysfunction and its relation to visceral obesity
    Original languageSpanish
    Pages (from-to)133-153
    Number of pages21
    JournalMedicina Interna de Mexico
    Volume21
    Issue number2
    StatePublished - Mar 1 2005

    Keywords

    • Endothelial dysfunction
    • Visceral obesity

    ASJC Scopus subject areas

    • Internal Medicine

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