Aspectos endocrinos, metabólicos y genéticos de la disfunción endotelial y su relación con la obesidad visceral

Raúl A. Bastarrachea Sosa; G. Castro Martínez; H. Laviada Molina; R. Yza Villanueva; G. Llamas Esperón; A. G. Comuzzie

Aspectos endocrinos, metabólicos y genéticos de la disfunción endotelial y su relación con la obesidad visceral

Translated title of the contribution: Endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation to visceral obesity

Raúl A. Bastarrachea Sosa, G. Castro Martínez, H. Laviada Molina, R. Yza Villanueva, G. Llamas Esperón, A. G. Comuzzie

Research output: Contribution to journal › Review article › peer-review

Abstract

Atherosclerosis is the combination of pathological changes that not only affect cells deposited along the walls of the arteries, but also the inflammatory ones. Stimulus implied in the cause of atherosclerosis, which include LDL, mechanical forces followed by high blood pressure, and inflammatory response induced by metabolic and hemodynamic aspects as risk factors, are diverse. Resistance to insulin, followed by visceral obesity and diabetes mellitus type 2 represent equally chronic and complex systemic alterations. Thus, it is not strange that the intersection of these disorders and their final outcomes cause atherosclerotic diabetes. This article reviews the endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation with visceral obesity.

Translated title of the contribution	Endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation to visceral obesity
Original language	Spanish
Pages (from-to)	133-153
Number of pages	21
Journal	Medicina Interna de Mexico
Volume	21
Issue number	2
State	Published - Mar 1 2005

Keywords

Endothelial dysfunction
Visceral obesity

ASJC Scopus subject areas

Internal Medicine

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Aspectos endocrinos, metabólicos y genéticos de la disfunción endotelial y su relación con la obesidad visceral. / Bastarrachea Sosa, Raúl A.; Castro Martínez, G.; Laviada Molina, H.; Yza Villanueva, R.; Llamas Esperón, G.; Comuzzie, A. G.

In: Medicina Interna de Mexico, Vol. 21, No. 2, 01.03.2005, p. 133-153.

Research output: Contribution to journal › Review article › peer-review

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abstract = "Atherosclerosis is the combination of pathological changes that not only affect cells deposited along the walls of the arteries, but also the inflammatory ones. Stimulus implied in the cause of atherosclerosis, which include LDL, mechanical forces followed by high blood pressure, and inflammatory response induced by metabolic and hemodynamic aspects as risk factors, are diverse. Resistance to insulin, followed by visceral obesity and diabetes mellitus type 2 represent equally chronic and complex systemic alterations. Thus, it is not strange that the intersection of these disorders and their final outcomes cause atherosclerotic diabetes. This article reviews the endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation with visceral obesity.",

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AU - Bastarrachea Sosa, Raúl A.

AU - Castro Martínez, G.

AU - Laviada Molina, H.

AU - Yza Villanueva, R.

AU - Llamas Esperón, G.

AU - Comuzzie, A. G.

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N2 - Atherosclerosis is the combination of pathological changes that not only affect cells deposited along the walls of the arteries, but also the inflammatory ones. Stimulus implied in the cause of atherosclerosis, which include LDL, mechanical forces followed by high blood pressure, and inflammatory response induced by metabolic and hemodynamic aspects as risk factors, are diverse. Resistance to insulin, followed by visceral obesity and diabetes mellitus type 2 represent equally chronic and complex systemic alterations. Thus, it is not strange that the intersection of these disorders and their final outcomes cause atherosclerotic diabetes. This article reviews the endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation with visceral obesity.

AB - Atherosclerosis is the combination of pathological changes that not only affect cells deposited along the walls of the arteries, but also the inflammatory ones. Stimulus implied in the cause of atherosclerosis, which include LDL, mechanical forces followed by high blood pressure, and inflammatory response induced by metabolic and hemodynamic aspects as risk factors, are diverse. Resistance to insulin, followed by visceral obesity and diabetes mellitus type 2 represent equally chronic and complex systemic alterations. Thus, it is not strange that the intersection of these disorders and their final outcomes cause atherosclerotic diabetes. This article reviews the endocrine, metabolic and genetic aspects of endothelial dysfunction and its relation with visceral obesity.

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Aspectos endocrinos, metabólicos y genéticos de la disfunción endotelial y su relación con la obesidad visceral

Abstract

Keywords

ASJC Scopus subject areas

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