A RIPtide Protects Neurons from Infection

Ryan P. Gilley; William J. Kaiser

doi:10.1016/j.chom.2017.03.014

A RIPtide Protects Neurons from Infection

Ryan P. Gilley, William J. Kaiser

Microbiology, Immunology & Molecular Genetics

Research output: Contribution to journal › Short survey › peer-review

2 Scopus citations

Abstract

RIPK3 and RIPK1 limit virus spread by executing either apoptotic or necroptotic cell death in response to infection. In a recent issue of Cell, Daniels et al. (2017) unveil an unexpected cell death-independent requirement of RIP kinase activity in coordinating neuroinflammation, restricting West Nile virus pathogenesis in neurons.

Original language	English (US)
Pages (from-to)	415-416
Number of pages	2
Journal	Cell Host and Microbe
Volume	21
Issue number	4
DOIs	https://doi.org/10.1016/j.chom.2017.03.014
State	Published - Apr 12 2017

ASJC Scopus subject areas

Parasitology
Microbiology
Virology

Access to Document

10.1016/j.chom.2017.03.014

Fingerprint Dive into the research topics of 'A RIPtide Protects Neurons from Infection'. Together they form a unique fingerprint.

Cite this

@article{0ce2e2740636494c9ee7132c32e44d87,

title = "A RIPtide Protects Neurons from Infection",

abstract = "RIPK3 and RIPK1 limit virus spread by executing either apoptotic or necroptotic cell death in response to infection. In a recent issue of Cell, Daniels et al. (2017) unveil an unexpected cell death-independent requirement of RIP kinase activity in coordinating neuroinflammation, restricting West Nile virus pathogenesis in neurons.",

author = "Gilley, {Ryan P.} and Kaiser, {William J.}",

year = "2017",

month = apr,

day = "12",

doi = "10.1016/j.chom.2017.03.014",

language = "English (US)",

volume = "21",

pages = "415--416",

journal = "Cell Host and Microbe",

issn = "1931-3128",

publisher = "Cell Press",

number = "4",

}

TY - JOUR

T1 - A RIPtide Protects Neurons from Infection

AU - Gilley, Ryan P.

AU - Kaiser, William J.

PY - 2017/4/12

Y1 - 2017/4/12

N2 - RIPK3 and RIPK1 limit virus spread by executing either apoptotic or necroptotic cell death in response to infection. In a recent issue of Cell, Daniels et al. (2017) unveil an unexpected cell death-independent requirement of RIP kinase activity in coordinating neuroinflammation, restricting West Nile virus pathogenesis in neurons.

AB - RIPK3 and RIPK1 limit virus spread by executing either apoptotic or necroptotic cell death in response to infection. In a recent issue of Cell, Daniels et al. (2017) unveil an unexpected cell death-independent requirement of RIP kinase activity in coordinating neuroinflammation, restricting West Nile virus pathogenesis in neurons.

UR - http://www.scopus.com/inward/record.url?scp=85017342925&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=85017342925&partnerID=8YFLogxK

U2 - 10.1016/j.chom.2017.03.014

DO - 10.1016/j.chom.2017.03.014

M3 - Short survey

C2 - 28407477

AN - SCOPUS:85017342925

VL - 21

SP - 415

EP - 416

JO - Cell Host and Microbe

JF - Cell Host and Microbe

SN - 1931-3128

IS - 4

ER -