Abstract
RIPK3 and RIPK1 limit virus spread by executing either apoptotic or necroptotic cell death in response to infection. In a recent issue of Cell, Daniels et al. (2017) unveil an unexpected cell death-independent requirement of RIP kinase activity in coordinating neuroinflammation, restricting West Nile virus pathogenesis in neurons.
Original language | English (US) |
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Pages (from-to) | 415-416 |
Number of pages | 2 |
Journal | Cell Host and Microbe |
Volume | 21 |
Issue number | 4 |
DOIs | |
State | Published - Apr 12 2017 |
ASJC Scopus subject areas
- Parasitology
- Microbiology
- Virology